CAV1 and hydrops fetalis: Given that eHFSCs critically participate in later stages of cutaneous wound healing (e.g., by producing progenitor cells that migrate out of the bulge into the epidermis to facilitate re-epithelialization [61,62,63]), and the fact that Cav1 downregulation is required for wound closure [55,56], the role of Cav1 might extend to epithelial HF progenitor cells and their emigration into the epidermis.