Although, in vitro, the resistance of some lamin A/C deficient cancer cells to apoptosis induced by pulses of shear stress was reported to be lower compared to control cells [49], in our experimental setting lamin A/C B16F10 cells accumulating in the lung vasculature shortly after injection did not experience elevated cell death, as indicated by their normal recovery from total lung cell suspensions (Figure 3D). The gene discussed is LMNA; the disease is cancer.