IL6 and COVID-19: The cytokine storm hypothesis is not able to explain the extrapulmonary damages produced by SARS-CoV-2 infection as the median levels of IL-6 in patients with severe COVID-19 are 10- to 200-fold smaller (see Table 1) than those observed in patients with hyperinflammatory phenotype of acute respiratory distress syndrome (ARDS) (see also the comments in References [33,34]):