Furthermore, this study denoted that Gal-3 expression could augment expression of antiviral genes including IFN-β, IFN-γ, PKR, and 2′,5′-OAS via regulating JAK/STAT pathways in the infected cells and proposed that Gal-3 exerted cytokine-like regulatory actions, demonstrating that Gal-3 expression has anti-influenza capabilities [38] (Figure 3). This evidence concerns the gene IFNG and influenza.