MALAT1 and cervical cancer: By performing secondary data analyses of dimethyl sulfate-sequencing (DMS-Seq) data from human erythroleukemic cell line K562 and psoralen analysis of RNA interactions and structure (PARIS) data from cervical cancer-derived HeLa cells compared to the working structural model of MALAT1 in noncancerous cells, the authors postulated that m6A-based structural changes of MALAT1 might mediate cancer in a cell-type-specific manner [107].