Consistent with previous studies that showed elevated serum and CSF levels of endothelial adhesion molecules in MMD [22], in the present study, ICAM1/2, CXCR1/2, and CCR6 were predicted and regulated, and there was also one pathway highlighted as “cell adhesion molecules” in the in-depth analyses of the DE-mRNA, implicating the transmigration process of leucocytes in aMMD. The gene discussed is CXCR1; the disease is multiminicore myopathy.