In addition, research has shown that CXCR2-mediated neutrophil recruitment is essential for lung fibrosis (Russo et al., 2009; Konrad and Reutershan, 2012; Liang et al., 2016; Hoegl et al., 2017), and that blocking CXCR2 can reduce lung fibrosis (Russo et al., 2009). The gene discussed is CXCR2; the disease is pulmonary fibrosis.