Li et al. found that miR-7a/b could improve cardiac function and inhibit MF and apoptosis after myocardial infarction mainly by regulating SP-1 and POLYadenosine diphosphate ribose polymerase-1 in a myocardial infarction mouse model (Li R. et al., 2016). Here, SP1 is linked to myocardial infarction.