showed that overexpression of lncRNA KCNQ1OT1 improved heart function in LPS-induced sepsis rat; lncRNA KCNQ1OT1 attenuated myocardial apoptosis and inflammatory factors by sponging miR-192-5p that targets X-chromosome-linked inhibitor of apoptosis (XIAP) in LPS-induced H9c2 cells (Sun et al., 2020). This evidence concerns the gene KCNQ1OT1 and Sepsis.