Pérez-Torras et al. (2013) found that the restoration of human CNT1 in pancreatic cancer cells caused an alteration in cell cycle progression and in the phosphorylation status of kinases involved in key signaling pathways, promoted polyADPR polymerase activity and non-apoptotic cell death, and decreased cell migration, thus reducing tumor growth. The gene discussed is SLC28A1; the disease is neoplasm.