Analogously, Belyk et al. suggested that polymorphisms in PLXNA1 are associated with altered developmental trajectory of the CC microstructure.36 Additionally, three patients had either signs of congenital cranial neuropathies including sensorineural hearing loss with or without agenesis of vestibulocochlear nerves (C:II-2, G:II-1), unilateral facial palsy (G:II-1), and peripheral axonal neuropathy (D:II-2). This evidence concerns the gene PLXNA1 and axonal neuropathy.