Since the slower alcohol metabolizing ADH1B TC/CC genotype is known to increase susceptibility to alcoholism [14–16] in a culture that encourages drinking after work-related functions, [14–16] a heavier intake may lead to high blood pressure, reduced platelet aggregation, and enhanced fibrinolysis, which may elevate the risk of hemorrhagic events [17]. The gene discussed is ADH1B; the disease is alcohol drinking.