NFKB1 and neoplasm: (64) proposed that VEGF production, in collaboration with NF-κB signaling in GC-MSC, could induce angiogenesis through driving the human umbilical vein endothelial cells (HUVEC) tube formation, suggesting the active involvement of GC-MSC in tumor neovascularization, while the inherent regulation mechanisms in VEGF/NF-κB signaling remains unquestioned.