WNT7B and diabetes mellitus: Although their expression levels have not been determined in islets of diabetic patients, some studies showed that Wnt7a and Wnt7b are required for pancreatic development through autocrine and paracrine mechanisms106, 107; therefore, stably expressing Wnt7a or Wnt7b alone could enhance the proliferation of human pancreatic progenitor cells (PPCs) through activating the non‐canonical Wnt/PKC signalling, suggesting their promising application in developing cell therapies for diabetes.107