The combination of bispecific antibodies, (Her2 x CD3) or (Her2 x Vγ2) (8, 9), together with the transferred Vγ2Vδ2 T cells in the presence of IL2, achieved a delay in the growth of pancreatic ductal adenocarcinoma tumor in murine models (10). Here, ERBB2 is linked to pancreatic ductal adenocarcinoma.