Thiel et al. found that miR-183 and miR-96 amounts are elevated in CD4+ T cells obtained from the peripheral blood of Graves' orbitopathy (GO) cases, and adoptive transfer of miR-183 and miR-96 overexpressing antigen-specific T cells accelerates the onset of autoimmune diabetes, whereas transferring specific antagomirs in CD4+ T cells prolongs disease onset [36]. The gene discussed is CD4; the disease is geroderma osteodysplastica.