To test whether oncogenic PI3Kα also mimicked oncogenic Kras on tumour/stroma interaction, we assessed F4/80+ macrophage recruitment and infiltrating CD206+‐macrophages around tumours and found them at a similar rate in peritumoural tissue from hyper‐activated oncogenic PI3Kα (p110αH1047R) and KRAS mutant tumours (Fig 7K and L). This evidence concerns the gene KRAS and neoplasm.