SOAT1 and colonic neoplasm: As a result, genes in the red module were mainly enriched into Autoimmune thyroid disease, Phagosome, and Cell adhesion molecules (CAMs) pathways; whereas genes in the purple module were mainly enriched into the JAK-STAT, Wnt, and T cell receptor signal pathways (Figures 2F,G), demonstrating that the resistance of colon cancer to oxaliplatin might be related to immunity and cell migration.