Results revealed that treatment with NaHS (100 μmol/kg/day, i.p., 4 weeks) obviously decreased the level of MDA, a lipoxidation product (Figure 6A), increases the level of GSH (Figure 6B) and the activity of endogenous antioxidant enzyme SOD (Figure 6C) in the hippocampal of CRS-treated rats. This evidence concerns the gene SOD1 and congenital rubella syndrome.