Between these two time points of postnatal development, H3K79me2 changes in control samples are associated to axon sprouting, mitochondrial activity, complement cascade and cerebral cortical regionalization with associated genes such as Pcdh18, identified as a potential candidate for intellectual disability (Kasnauskiene et al., 2012; Supplementary Figure 5D). The gene discussed is PCDH18; the disease is Intellectual disability.