AGT and pulmonary edema: Downregulation of such receptors after being attached to the S protein of SARS-CoV-2 increases angiotensin II with excessive rise in blood pressure that can be attributable for both ischemic and hemorrhagic strokes development and increased bradykinins causes inflammatory states in the lungs with hypoxemia that may further complicate cerebrovascular insults also increase in angiotensin II production increases vascular permeability to the degree of causing pulmonary edema [9].