Aberrant T cell activation and proliferation are one of the mechanisms responsible for IBD, where high amounts of pro-inflammatory cytokines such as tumor necrosis factor-α (TNF-α), interleukin-17 (IL-17), or interferon-γ (IFN-γ) are released in the gut epithelium by activated Th1, or Th17 [3]. This evidence concerns the gene IL17A and inflammatory bowel disease.