Whether H pylori infection delays gastric emptying is unclear,[13,14] but H pylori appears to alter gastric acid production by changing gastrin and somatostatin secretion.[15] Abnormal gastric acid secretion causes mainly dysmotility-like, dyspeptic symptoms.[16] Duodenal acid exposure indirectly induces fullness, bloating, and epigastric pain by suppressing antral contractions, which may contribute to delayed gastric emptying.[17] Previous studies on the effect of H pylori eradication on FD are conflicting. This evidence concerns the gene SST and Fabry disease.