SLC2A1 and epilepsy: Disappointingly,SLC2A1/GLUT1 abundance did not correlate with [18F]-activity,possibly because glucose delivery may not be the rate-determiningstep for its utilization by epileptogenic tissue.20,21,41 In addition, low spatial resolution andspill-in within volumes of cubic millimeters might not capture potentiallyaltered function of aberrant blood vessels that characterize chronic,drug-resistant epilepsy.