SOCS1 and asthma: MiR-19a expression is dramatically raised in T lymphocytes infiltrated in the airway of asthma, and miR-19a can regulate PTEN, SOCS1 and A20 to cooperatively induce the production of Th2 cytokines [29]; miR-142-3p takes part in regulating the multiplication and differentiation of airway smooth muscle cells in asthma by inhibiting WNT signaling pathway [30]; miR-149 overexpression can inhibit inflammatory cytokines’ expressions in HSAECs induced by PAF, and alleviate the barrier damage of epithelial cells [31].