GO analyses showed that multiple immune-related processes were enriched in TERTmut gliomas, including cytokine-cytokine receptor interaction, chemokine signaling pathway, graft-versus-host disease, intestinal immune network for IgA production, Jak-STAT signaling pathway, antigen processing and presentation, HTLV-I infection, NF-κB signaling pathway, natural killer cell-mediated cytotoxicity, and PI3K-Akt signaling pathway (Figure 2B), which verified our hypothesis that TERT promoter status is associated with immune response in glioma. This evidence concerns the gene CD79A and graft versus host disease.