Besides, it is noteworthy that while in most cases the inhibition of HDAC or HMTs resulted in repression of EMT and tumor growth, in some contradictory studies it showed precisely the opposite effect, again highlighting the context-dependent behaviors of epigenetic-regulated EMT networks and implying their possible role as MET blockers at the late stages of metastasis (86–88). The gene discussed is MET; the disease is neoplasm.