Ginsenoside Rh7 can regulate the expression of ILF3-AS1, ILF3-AS1 competitively binds with miR-212 in turn, and the miR-212-Smad1 axis could be enhanced by ginsenoside Rh7, thus ginsenoside Rh7 could significantly inhibit the proliferation, migration and invasion of NSCLC cells (Figure 10). Here, SMAD1 is linked to non-small cell lung carcinoma.