Aβ was shown to be an AMP with as potent activity as LL-37 cathelicidin (Soscia et al., 2010; Gosztyla et al., 2018) acting against various bacteria, fungi, and viruses supporting the “Pathogen Hypothesis” of AD according to which cerebral infections cause the AD (Robinson et al., 2004). Here, CAMP is linked to Alzheimer disease.