A total of nine KEGG pathways were significantly enriched (p < 0.05, Fig. 4) including those for neuroactive ligand-receptor interaction, complement and coagulation cascades, Staphylococcus aureus infection, ovarian steroidogenesis, prolactin signaling pathway, PI3K − Akt signaling pathway, cAMP signaling pathway, GnRH signaling pathway, and inflammatory mediator regulation of TRP channels. This evidence concerns the gene GNRH1 and staphylococcus aureus infection.