Gallic acid, a major chemical component of QLX, was reported to inhibit NF‐κB expression and activation followed by down‐regulation of IL‐1, ILI‐6, IL‐12, IL‐17 and IL‐23, TGF‐β and TNF‐α expression in ulcerative colitis.40 This evidence concerns the gene NFKB1 and ulcerative colitis.