For example, overexpression of the EGFR gene or protein kinase cascades downstream the growth factor receptor mutations, which were detected in a variety of solid tumors, in diverse acute myeloid leukemia (AML) cells, and in many other leukemic cells, highlighted their role as therapeutic targets and the importance of novel therapeutic approaches and treatment alternatives using NDDSs [1]. The gene discussed is EGFR; the disease is acute myeloid leukemia.