ACE2 and COVID-19: Neurological injury results from the affinity of the COVID-19 etiological agent, the Severe Acute Respiratory Syndrome coronavirus 2 (SARS-CoV-2), for the angiotensin-converting enzyme 2 (ACE2) receptor present in neurons and glial cells endowing high neuroinvasive potential to SARS-CoV-2 compared to previous coronaviruses.