Based upon the expression levels of these miRNAs, we found that miR‐149* (−3p) was significantly upregulated (identified as protective) in GMLs and found to be associated with genes involved in T‐cell proliferation, inhibition of apoptosis by targeting PD‐1, TIM‐3, BTLA, Foxp1, IL‐2, TNF‐α, and IFN‐γ in breast cancer cells.48 The gene discussed is BTLA; the disease is breast carcinoma.