Flow cytometry experiments revealed that METTL3 restoration partly eliminated apoptosis's promoting effects, induced due to BLACAT2-knockdown in both AGS and MGC-803 cells (Figure 7D). Plate colony formation assay showed that the METTL3 restoration partly reversed the suppressive effects of proliferation induced by BLACAT2-knockdown in both AGS and MGC-803 cells (Supplementary Figure 4C-D). In a nutshell, these data proved that BLACAT2 promotes gastric cancer progression by upregulating the expression of METTL3, which inhibits miR-193b-5p. The gene discussed is METTL3; the disease is gastric cancer.