LINC00649 and gastric cancer: Specifically, our data evidenced that LINC00649 tended to be enriched in GC tissues and cells, in contrast with the normal counterparts, and LINC00649 positively regulated GC cell proliferation, migration, epithelial-mesenchymal transition (EMT) and tumorigenesis in vitro and in vivo, hinting that LINC00649 served as an oncogene for GC.