Infection with BRSV leads to the initiation of the inflammatory cytokine response, with increases in several cytokines including IFNγ, IL-12β, IL-6, TNF, IL-18, CXCL8, CCL3, CCL5, CCL2, IFNα1, and IFNβ1, and the subsequent influx of leukocytes, predominantly neutrophils, produces inflammation and lung pathology (Valarcher and Taylor, 2007; Sudaryatma et al., 2018). This evidence concerns the gene IFNG and infection.