In the present study, similar to SBM, enteritis induced by FSBM was enriched in immune-related signaling pathways, such as NF-κB signaling pathway, TNF signaling pathway, antigen processing and presentation, intestinal immune network producing IgA, inflammatory bowel disease (IBD), B cell receptor, and JAK-STAT. The gene discussed is SOAT1; the disease is inflammatory bowel disease.