The influence on rhodopsin in Adipor1 KO mice was also in contrast to ciliopathy, a congenital disease in which no POSs develop, and associated with ectopic rhodopsin expression due to impaired rhodopsin trafficking33; Adipor1 KO mice, which also showed POS deficiency, did not exhibit ectopic rhodopsin expression and simply showed rhodopsin repression. The gene discussed is ADIPOR1; the disease is ciliopathy.