TGFB1 and liver dysplastic nodule: In STZ-induced diabetic mice, AR deficiency significantly increases the miR-200a-3p/miR-141-3p levels in the renal cortex, which is accompanied by the significant downregulation of Keap1, Tgfβ1/2, and fn1, and the prominent upregulation of Nrf2. Therefore, the inhibition of AR and the restoration of the miR-200a-3p/miR-141-3p levels may be a potential research direction for the treatment of DN.