When comparing against mHALT-1 and anti-mKRAS G12V-34 scFv standalone controls (Figs. 8C & 8D), the mHALT-1-scFv immunotoxin was shown to re-exhibit its cytotoxicity effects on KRAS-positive cancer cells with minimal killing of KRAS-negative cells, thus reinforcing the notion that the scFv moiety acted as a targeting tool for the mHALT-1 toxin. The gene discussed is KRAS; the disease is cancer.