However, activation of the NF-κB signaling pathway would lead to the induction of essential proinflammatory molecules for the host antipathogen response [46, 47], although previous studies have shown that the NF-κB signaling limited the apoptosis of infected cells to facilitate the growth and maturation of C. parvum at the early infection stage [31, 48]. The gene discussed is NFKB1; the disease is infection.