CASP3 and coronary artery disorder: It was very recently showed that miR-98 levels were obviously decreased in single-, double-, and multivessel lesion CAD patients and 24-hour hypoxic-induced HUVEC, while ago-miR-98 remarkably suppressed caspase-3 activity, reduced atherosclerosis, and markedly increased cellular viability through targeting LOX-1, which may be used for possible molecular drug targets for atherosclerotic CAD subjects [84].