Furthermore, a recent mouse study indicated that bronchopulmonary dysplasia (BPD), a severe complication of the respiratory system seen in preterm infants, was induced by IL-33/ILC2 signaling caused by an arrest in the process of alveolarization, determining a major destructive role of tissue-resident IL-13+ILC2 in the lung (36). This evidence concerns the gene IL13 and bronchopulmonary dysplasia.