On the contrary, other novel deletions were either described in other tumors, such as CDH11 in retinoblastoma [78] or ROBO1 in breast cancer [79]; or affected genes vinculated with cancer pathways such as the tumor suppressor & non-coding RNA LINC00472 [80], the B-cell specific maturation regulator TNFRSF13C (BAFF receptor) [81], the immune checkpoint PVR [82], the receptor tyrosine kinase-coupled signaling regulator SIRPB1 [83] or the proapoptotic gene APAF1 [84]. This evidence concerns the gene APAF1 and breast carcinoma.