To elucidate which substrates are key for this process and evaluate a potential role of fatty acid (FA) in the survival of MRD-mimic cancer cells, we first blocked fatty acid oxidation (FAO) using etomoxir, an inhibitor of the carnitine palmitoyl transferase (CPT1) that imports FA into mitochondria for β-oxidation (25). The gene discussed is CPT1A; the disease is cancer.