We demonstrated that pharmacological activation of BAT increases LPL‐mediated TG‐derived FA uptake by interscapular BAT, subscapular BAT, and even perivascular adipose tissue (pVAT) that has characteristics of both WAT and BAT; these effects are accompanied by enhanced hepatic TRL‐core remnant clearance, thereby attenuating dyslipidemia, and thus atherosclerosis development (Berbee et al., 2015). This evidence concerns the gene LPL and metabolic syndrome.