RES promotes the epithelial-type alternative splicing of Cd44, Enah, and FGFR2 pre-mRNAs in breast cancer cells by upregulating KH-type splicing regulatory protein (KHSRP) and heterogeneous nuclear ribonucleoproteins A1 (hnRNPA1) expression (33). This evidence concerns the gene KHSRP and breast cancer.