The main enrichment pathways for the sample genes from the TNFRSF12A high expression group were bladder cancer, cell adhesion molecule cams, cytokine-cytokine receptor interaction, ECM receptor interaction, focal adhesion, hematopoietic cell lineage, the JAK/STAT signaling pathway, leukocyte transendothelial migration, natural killer cell-mediated cytotoxicity, and the Toll-like-receptor signaling pathway. This evidence concerns the gene SOAT1 and urinary bladder cancer.