The overexpressed lncRNA UCA1 increased the expression levels of key molecules in the PI3K/AKT/mTOR signaling pathway, including AKT serine/threonine kinase 3 (AKT3), phosphorylated mammalian target of rapamycin (p-mTOR), and ribosomal protein S6 kinase (S6K), while reducing the eukaryotic translation initiation factor 4E (EIF4E) protein levels in GC cells [49]. This evidence concerns the gene MTOR and gastric cancer.